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parametric pearson chi square tests  (CH Instruments)

 
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    CH Instruments parametric pearson chi square tests
    Parametric Pearson Chi Square Tests, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/parametric pearson chi square tests/product/CH Instruments
    Average 90 stars, based on 1 article reviews
    parametric pearson chi square tests - by Bioz Stars, 2026-03
    90/100 stars

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    AKT activation correlates with p53 mutation in triple-negative breast cancers of obese patients. (A) Immunostaining with antibodies against p53 and p-AKT S473 of representative triple-negative breast cancer sections derived from obese and nonobese patients. H, hematoxylin staining. (Scale bars: 100 μm.) (B) Graphs summarize p-AKT S473 staining in tumor samples from 32 obese patients [body mass index (BMI) > 30] and 20 nonobese patients, sorted according to p53 status. The horizontal bar indicates the median. Correlations were analyzed by a nonparametric Mann–Whitney U test. (C) Table summarizes the same tumor samples as in B, grouped according to p53 status and p-AKT S473 staining. Tumors with p53 staining >80% have been classified as mutant p53. Tumors with p-AKT staining >15% have been classified as “positive.” Correlations were analyzed by a parametric <t>Pearson’s</t> chi-square test (P-val). (D) Proposed model for the gain of function of mutant p53 in the response to insulin as mediated by its cytoplasmic interaction with DAB2IP.
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    Average 90 stars, based on 1 article reviews
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    AKT activation correlates with p53 mutation in triple-negative breast cancers of obese patients. (A) Immunostaining with antibodies against p53 and p-AKT S473 of representative triple-negative breast cancer sections derived from obese and nonobese patients. H, hematoxylin staining. (Scale bars: 100 μm.) (B) Graphs summarize p-AKT S473 staining in tumor samples from 32 obese patients [body mass index (BMI) > 30] and 20 nonobese patients, sorted according to p53 status. The horizontal bar indicates the median. Correlations were analyzed by a nonparametric Mann–Whitney U test. (C) Table summarizes the same tumor samples as in B, grouped according to p53 status and p-AKT S473 staining. Tumors with p53 staining >80% have been classified as mutant p53. Tumors with p-AKT staining >15% have been classified as “positive.” Correlations were analyzed by a parametric Pearson’s chi-square test (P-val). (D) Proposed model for the gain of function of mutant p53 in the response to insulin as mediated by its cytoplasmic interaction with DAB2IP.

    Journal: Proceedings of the National Academy of Sciences of the United States of America

    Article Title: Mutant p53 potentiates the oncogenic effects of insulin by inhibiting the tumor suppressor DAB2IP

    doi: 10.1073/pnas.1700996114

    Figure Lengend Snippet: AKT activation correlates with p53 mutation in triple-negative breast cancers of obese patients. (A) Immunostaining with antibodies against p53 and p-AKT S473 of representative triple-negative breast cancer sections derived from obese and nonobese patients. H, hematoxylin staining. (Scale bars: 100 μm.) (B) Graphs summarize p-AKT S473 staining in tumor samples from 32 obese patients [body mass index (BMI) > 30] and 20 nonobese patients, sorted according to p53 status. The horizontal bar indicates the median. Correlations were analyzed by a nonparametric Mann–Whitney U test. (C) Table summarizes the same tumor samples as in B, grouped according to p53 status and p-AKT S473 staining. Tumors with p53 staining >80% have been classified as mutant p53. Tumors with p-AKT staining >15% have been classified as “positive.” Correlations were analyzed by a parametric Pearson’s chi-square test (P-val). (D) Proposed model for the gain of function of mutant p53 in the response to insulin as mediated by its cytoplasmic interaction with DAB2IP.

    Article Snippet: Tumors with p-AKT staining >15% have been classified as “positive.” Correlations were analyzed by a parametric Pearson’s chi-square test (P-val). ( D ) Proposed model for the gain of function of mutant p53 in the response to insulin as mediated by its cytoplasmic interaction with DAB2IP.

    Techniques: Activation Assay, Mutagenesis, Immunostaining, Derivative Assay, Staining, MANN-WHITNEY